Ubiquitination in NAFLD: Regulation of Lipid Accumulation, Inflammation, and Fibrosis
The development of NAFLD involves intricate molecular mechanisms that encompass several pathways working in tandem. Ubiquitination plays a crucial role in regulating the classical targets that contribute to lipid accumulation, inflammation, and fibrosis, such as Sterol Regulatory Element-Binding Protein 1C (SREBP1c), Nuclear Factor Kappa-B (NF-κB), and Transforming Growth Factor-Beta (TGF-β) pathway.
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