在机制上PTL阻断了EGF诱导的FAK、GSK3β蛋白的磷酸化进一步研究发现PTL能通过抑制由EGF介导的FAKGSK3β信号通路的磷酸化来抑制宫颈癌细胞的增殖。总之我们的研究阐明了PTL可能是通过调控FAKGSK3β信号通路来抑制宫颈癌细胞的增殖和迁移能够为PTL在宫颈癌治疗领域的应用提供线索。在医学SCI期刊论文中有哪些英语表述？

"In terms of mechanism, PTL inhibited the phosphorylation of FAK and GSK3β proteins induced by EGF. Further investigations revealed that PTL could suppress the proliferation of cervical cancer cells by inhibiting the phosphorylation of the FAK/GSK3β signaling pathway mediated by EGF. In summary, our study elucidates that PTL may inhibit the proliferation and migration of cervical cancer cells by regulating the FAK/GSK3β signaling pathway, providing clues for the application of PTL in the treatment of cervical cancer."
"Our findings demonstrate that PTL exerts its anti-proliferative and anti-migratory effects on cervical cancer cells by modulating the FAK/GSK3β signaling pathway, which is mediated by EGF. Therefore, PTL may serve as a potential therapeutic agent for cervical cancer."
"The present study highlights the importance of the FAK/GSK3β signaling pathway in the pathogenesis of cervical cancer, and suggests that PTL could be a promising candidate for the treatment of this disease.